ECG findings in Acute Pericarditis

ECG findings in Acute Pericarditis

EKG findings suggestive of acute pericarditisacute pericarditisMyocardial Infarctiom
1ST-elevation is less than 5 mm
2ST-segment concavityST-segment elevation usually “concave” upward
3More extensive lead involvementDiffuse ST elevation ST-elevation related to location of ischemia  
4Less prominent reciprocal ST-segment depression
5PR-segment elevation in aVR, with reciprocal PR-segment depression in other leadsPR-segment depression often occursNo PR-segment depression  
6The absence of abnormal Q-waves
7Variability in the time of T-wave inversion occurrence following ST-segment elevation
8The lack of QRS widening and QT interval shortening in leads with ST-elevation

Spodick’s sign

Spodick’s sign refers to a downsloping TP segment, best visualized in lead II and lateral precordial leads

ECG findings in Acute Pericarditis – MCQs


1. Which of the following is the most characteristic ECG feature of acute pericarditis?
ST depression in all leads
Peaked T waves
Diffuse ST elevation with PR depression
Q waves in lateral leads
In acute pericarditis, diffuse ST elevation with PR segment depression is a hallmark ECG feature, especially in limb and precordial leads.

2. In acute pericarditis, which ECG phase shows normalization of ST segments?
Phase I
Phase III
Phase IV
Phase II
Phase II is the stage when ST segments begin to normalize following the initial diffuse ST elevation and PR depression of early stages.

3. PR segment depression in ECG is most prominently seen in which leads?
Inferior leads (II, III, aVF)
aVR only
Lead V1 only
Lateral leads
PR segment depression is typically most noticeable in inferior leads due to inflammation of the atrial epicardium.

4. What ECG change helps to distinguish acute pericarditis from early repolarization?
Presence of PR segment depression
ST elevation in precordial leads
Notched J point
T wave inversion
PR segment depression is not a feature of early repolarization, helping distinguish it from acute pericarditis.

5. Which ECG lead commonly shows reciprocal changes (e.g., PR elevation) in acute pericarditis?
aVR
V5
II
V3
Lead aVR often shows reciprocal changes such as PR segment elevation and ST depression in acute pericarditis.

6. What best describes T wave changes in the evolution of acute pericarditis?
T waves remain normal
T wave inversion occurs after ST segments normalize
Early T wave inversion
Tall symmetrical T waves in V1–V6
In acute pericarditis, T wave inversion typically occurs late, after ST segment resolution in Phase III.

7. What is typically absent in the ECG of acute pericarditis?
ST elevation
Pathological Q waves
PR depression
T wave inversion
Pathological Q waves are not typically seen in acute pericarditis; their presence should prompt evaluation for myocardial infarction.

8. Which phase of acute pericarditis ECG evolution includes widespread T wave inversion?
Phase I
Phase II
Phase IV
Phase III
Widespread T wave inversion typically occurs in Phase IV of acute pericarditis, as the final stage of ECG changes.

9. What ECG finding differentiates acute pericarditis from STEMI?
Diffuse rather than localized ST elevation
T wave inversion
Pathologic Q waves
Reciprocal ST depression
ST elevation in pericarditis is diffuse (across many leads), while in STEMI it is localized to the region of infarction.

10. What causes PR segment depression in acute pericarditis?
Atrial epicardial inflammation
Ventricular ischemia
Bundle branch block
P wave delay
PR segment depression occurs due to inflammation of the atrial epicardium, a distinguishing ECG feature in acute pericarditis.

11. What is the typical morphology of ST elevation in acute pericarditis?
Concave upwards
Convex downwards
Horizontal
Notched
The ST elevation in pericarditis typically has a concave upward (“saddle-shaped”) appearance.

12. Which of the following features is not typical of acute pericarditis ECG?
Widespread ST elevation
ST elevation in a single coronary artery territory
PR depression
Lack of reciprocal ST depression
Pericarditis typically causes diffuse ST elevation, not localized to a coronary artery territory like STEMI.

13. What is the earliest ECG change seen in acute pericarditis?
T wave inversion
Diffuse ST elevation and PR depression
Q waves
QT prolongation
The earliest ECG changes in acute pericarditis are widespread ST elevation and PR segment depression.

14. How long do the ECG changes in acute pericarditis typically evolve over?
Days to weeks
Minutes
Seconds
Hours
ECG changes in acute pericarditis typically evolve over a few days to weeks, in contrast to rapid evolution in MI.

15. In which condition is PR segment elevation most commonly seen?
Acute pericarditis
STEMI
Hyperkalemia
AV block
PR segment elevation in lead aVR (and reciprocal depression in other leads) is characteristic of acute pericarditis.

16. Which phase of pericarditis ECG shows normalization of ST segments?
Phase I
Phase III
Phase IV
Phase II
ST segments normalize during Phase II of pericarditis ECG evolution.

17. Which is the most common cause of ECG findings resembling acute pericarditis?
Viral infection
Myocardial infarction
Pulmonary embolism
Aortic dissection
Most cases of acute pericarditis (and its ECG changes) are due to viral infections.

18. What other condition can cause similar ST elevation but is usually localized?
ST-elevation myocardial infarction (STEMI)
Left bundle branch block
Right bundle branch block
Hyperthyroidism
STEMI also causes ST elevation but it is regional and not diffuse like in pericarditis.

19. What is the final ECG phase in acute pericarditis evolution?
Normalization
Persistent T inversion
ST depression
Q waves
Phase IV in ECG evolution of acute pericarditis ends with complete normalization of changes.

20. What best differentiates pericarditis ST elevation from early repolarization?
Associated PR depression
ST elevation in V2–V5
No QRS changes
T wave upright
PR segment depression is a hallmark of acute pericarditis and helps distinguish it from benign early repolarization.


ECG findings of Acute Pericarditis

Short-Answer Questions


1. What are the typical ECG changes seen in Stage 1 of acute pericarditis?
– Diffuse ST-segment elevation
– PR segment depression
– Upright T waves
– No reciprocal ST depression (except aVR, V1)
– No Q waves


2. How can acute pericarditis be distinguished from STEMI on ECG?
– ST elevation is diffuse, not localized to a coronary artery territory
– PR segment depression favors pericarditis
– No reciprocal ST depression except in aVR and V1
– No Q waves in pericarditis
– ST/T ratio > 0.25 in pericarditis


3. Describe the four ECG stages of acute pericarditis.
– Stage 1: ST elevation + PR depression
– Stage 2: Normalization of ST and PR
– Stage 3: T wave inversion
– Stage 4: ECG returns to baseline
– Changes usually occur over days to weeks


4. What is the PR segment abnormality seen in pericarditis?
– PR depression in multiple leads
– PR elevation in aVR and V1
– Caused by atrial epicardial inflammation
– Helps differentiate from STEMI
– Resolves as inflammation subsides


5. Why is the ST elevation in pericarditis called “concave upward”?
– ST segment curves upward like a smile
– Unlike convex (“tombstone”) ST in STEMI
– Suggests non-transmural inflammation
– Diffuse and benign-appearing pattern
– A hallmark of Stage 1 pericarditis


6. How does pericarditis affect the T wave over time?
– T waves are upright in early stages
– Become inverted in Stage 3
– Follow ST segment normalization
– Do not correlate with ischemia
– Eventually return to baseline


7. What is the significance of the aVR lead in pericarditis?
– Shows reciprocal PR elevation
– May show ST depression
– A clue to diffuse inflammation
– Helps distinguish from ischemia
– Not a primary diagnostic lead


8. Which leads typically show the most prominent changes in pericarditis?
– I, II, V5, V6
– Limb and lateral precordial leads
– Reflect pericardial surface inflammation
– ST elevation and PR depression evident
– Diffuse distribution pattern


9. What ECG finding suggests pericardial effusion rather than pericarditis?
– Low voltage QRS complexes
– Electrical alternans (if tamponade)
– No ST elevation or PR depression
– Alternating QRS amplitude
– Indicates swinging of heart in fluid


10. When does normalization of ECG occur in pericarditis?
– Over days to weeks
– Stage 2 shows initial resolution
– Stage 3 has T wave inversion
– Stage 4 is complete normalization
– Depends on inflammation control


FeatureAcute PericarditisNotes
Stage 1 (Early)Diffuse ST elevation (concave) + PR depressionMost diagnostic stage
Stage 2ST and PR segments normalizeFew days after onset
Stage 3T wave inversion beginsOccurs after ST normalization
Stage 4 (Late)ECG returns to baselineUsually within 2–3 weeks
ST Segment MorphologyConcave upward (“smiling”) ST elevationDiffers from convex ST in STEMI
Distribution of ST ElevationDiffuse (I, II, aVL, V3–V6)Not limited to a coronary artery territory
PR Segment ChangesPR depression in most leads; PR elevation in aVR, V1Reflects atrial epicardial inflammation
T Wave ChangesUpright in early stages; inversion in stage 3Does not indicate ischemia
Q WavesAbsentHelps distinguish from STEMI
Reciprocal ST DepressionTypically absent (except in aVR, V1)A clue against STEMI
Lead aVRPR elevation ± ST depressionReciprocal to pericarditis changes elsewhere
Electrical AlternansNot typical unless pericardial effusion/tamponade presentSuggests effusion > pericarditis
ST/T Ratio in Lead V6> 0.25Useful quantitative clue
Evolution TimelineStages evolve over days to weeksIn contrast to rapid changes in STEMI
Response to NSAIDsECG improves with anti-inflammatory therapyDiagnostic and therapeutic implication

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